YouSaid · the spoken record
Kristen Fortney
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- 2023-02-03
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- 2023-02-03
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“And that really excited me in particular because aging is so complicated, because aging is so difficult to intervene in, I thought that if we can learn what these people who are already aging successfully, what's different about them, that could be a really great way to figure out new drug targets, genetics has been really important in other areas of disease biology with PCSK9, et cetera, right? So I wanted to crack at those data. And so I joined the lab, got into that. And over time, I'd been realizing that what I really wanted to do was develop therapies and, you know, impact lives, impact health, right? And realizing more and more that that's not by and large what happens within academia, I mean, not for most people in academia.”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source
“Yeah, this is 2012, and I came to an aging biology lab at Stanford. And what attracted me in particular was that they had samples from people who lived to be like DNA samples, who lived to be over 100 or over 110, right?”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source
“Oh, definitely. It's a huge shift, right? Came in with lots of unrealistic ideas, I think, about how easy it was. And then you sort of meet all the hard realities of biology is actually really hard, right? And we still know so little. There's still so much to learn. And then, you know, I started to become more fluent in aging biology and aging science and decided for my postdoc, I really wanted to join like an aging focused lab. There are still very few of those”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source
“You know, I was always reading about aging biology. I was always very excited about that in particular because it seemed like it could have such tremendous potential for medicine, for healthcare, and it was still very new science. So I was always kind of like really reading from the outside. And as an undergraduate, I actually went into math and physics first, you know, but I was always kind of reading about aging biology on the side. And then for my PhD, I decided that I wanted to get into the field myself. So I did a PhD in bioinformatics, but focused on aging biology because I was sort of the quantitative skills that I knew applied to the problems that I cared about. And at the time, I analyzed large data sets to build biomarkers and to repurpose drugs. And really, I was like brand new to modern biology, so sort of really soaking all that up.”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source
“That really dates back an incredibly long time. I've been really fascinated by science and science fiction and aging biology since I was a kid, really, you know, probably reading tons of science fiction as a kid.”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source
“Sharing an awesome episode from our sister podcast, Bioeeds World, on the biology of aging. I know there can be a lot of judgment surrounding the world of longevity, but it's hard to think of a more potentially impactful space on humanity. Living longer might mean having the time to write that book you've always wanted to, to found that company you've always wanted to build, or just spend more time with your loved ones. But also, imagine having another decade to contribute at your highest potential. Imagine what you could do. Imagine how that could reshape society. And so naturally, the question becomes, what if you knew what markers to look for in order to extend life? Well, in this episode, A16Z General Partner Vijay Ponde and co-founder and CEO of BioHristin Fortney discuss exactly that. As a listener of this episode,”
2023-02-03 · a16z Podcast · Healthspan, Lifespan, and the Biology of Aging · IDENTIFIED FROM THE TRANSCRIPT · source